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physiostat ecg analysis software version 3.1  (Data Sciences International)

 
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    Data Sciences International physiostat ecg analysis software version 3.1
    M-mode <t> echocardiogram </t> before surgery on age-matched Clcn3 +/+ , Clcn3 +/− , and Clcn3 -/− mice.
    Physiostat Ecg Analysis Software Version 3.1, supplied by Data Sciences International, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/physiostat ecg analysis software version 3.1/product/Data Sciences International
    Average 90 stars, based on 1 article reviews
    physiostat ecg analysis software version 3.1 - by Bioz Stars, 2026-06
    90/100 stars

    Images

    1) Product Images from "Activation of Volume Regulated Chloride Channels Protects Myocardium from Ischemia/reperfusion Damage in Second-window Ischemic Preconditioning"

    Article Title: Activation of Volume Regulated Chloride Channels Protects Myocardium from Ischemia/reperfusion Damage in Second-window Ischemic Preconditioning

    Journal: Cellular Physiology and Biochemistry

    doi: 10.1159/000335858

    M-mode echocardiogram before surgery on age-matched Clcn3 +/+ , Clcn3 +/− , and Clcn3 -/− mice.
    Figure Legend Snippet: M-mode echocardiogram before surgery on age-matched Clcn3 +/+ , Clcn3 +/− , and Clcn3 -/− mice.

    Techniques Used:



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    (A–F) EpA960/MCM and MCM littermates were given tamoxifen (TAM) at the same time. (A and D) H&E stain revealed that EpA960/MCM hearts were dilated compared with hearts of tamoxifen-treated MCM mice. Original magnification, ×2. (B and E) H&E stain revealed that induced EpA960/MCM hearts exhibited hypertrophied cardiomyocytes that focally contained pale, granular cytoplasm compared with MCM hearts. Original magnification, ×40. (C and F) Electron microscopy revealed hypertrophied myocytes with irregular nuclei (N) and abundant mitochondria (m) in EpA960/MCM hearts compared with MCM hearts. Original magnification, ×4,000. (G) Cardiac functional abnormalities in mice expressing EpA960(R) mRNA. Doppler ultrasound was performed before (Pre) and after (Post) tamoxifen administration on EpA960/MCM and MCM littermates and revealed both systolic and diastolic dysfunction in hearts expressing EpA960(R) mRNA. (H) <t>ECG</t> telemetry revealed progressive arrhythmias following induction of expanded CUG RNA expression. The length of the PR interval (thick bar) is indicated; thin bars represent 100 ms.
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    (A–F) EpA960/MCM and MCM littermates were given tamoxifen (TAM) at the same time. (A and D) H&E stain revealed that EpA960/MCM hearts were dilated compared with hearts of tamoxifen-treated MCM mice. Original magnification, ×2. (B and E) H&E stain revealed that induced EpA960/MCM hearts exhibited hypertrophied cardiomyocytes that focally contained pale, granular cytoplasm compared with MCM hearts. Original magnification, ×40. (C and F) Electron microscopy revealed hypertrophied myocytes with irregular nuclei (N) and abundant mitochondria (m) in EpA960/MCM hearts compared with MCM hearts. Original magnification, ×4,000. (G) Cardiac functional abnormalities in mice expressing EpA960(R) mRNA. Doppler ultrasound was performed before (Pre) and after (Post) tamoxifen administration on EpA960/MCM and MCM littermates and revealed both systolic and diastolic dysfunction in hearts expressing EpA960(R) mRNA. (H) <t>ECG</t> telemetry revealed progressive arrhythmias following induction of expanded CUG RNA expression. The length of the PR interval (thick bar) is indicated; thin bars represent 100 ms.
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    (A–F) EpA960/MCM and MCM littermates were given tamoxifen (TAM) at the same time. (A and D) H&E stain revealed that EpA960/MCM hearts were dilated compared with hearts of tamoxifen-treated MCM mice. Original magnification, ×2. (B and E) H&E stain revealed that induced EpA960/MCM hearts exhibited hypertrophied cardiomyocytes that focally contained pale, granular cytoplasm compared with MCM hearts. Original magnification, ×40. (C and F) Electron microscopy revealed hypertrophied myocytes with irregular nuclei (N) and abundant mitochondria (m) in EpA960/MCM hearts compared with MCM hearts. Original magnification, ×4,000. (G) Cardiac functional abnormalities in mice expressing EpA960(R) mRNA. Doppler ultrasound was performed before (Pre) and after (Post) tamoxifen administration on EpA960/MCM and MCM littermates and revealed both systolic and diastolic dysfunction in hearts expressing EpA960(R) mRNA. (H) <t>ECG</t> telemetry revealed progressive arrhythmias following induction of expanded CUG RNA expression. The length of the PR interval (thick bar) is indicated; thin bars represent 100 ms.
    Physiostat Ecg Analysis Diagnostic Software, supplied by Data Sciences International, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Image Search Results


    M-mode echocardiogram before surgery on age-matched Clcn3 +/+ , Clcn3 +/− , and Clcn3 -/− mice.

    Journal: Cellular Physiology and Biochemistry

    Article Title: Activation of Volume Regulated Chloride Channels Protects Myocardium from Ischemia/reperfusion Damage in Second-window Ischemic Preconditioning

    doi: 10.1159/000335858

    Figure Lengend Snippet: M-mode echocardiogram before surgery on age-matched Clcn3 +/+ , Clcn3 +/− , and Clcn3 -/− mice.

    Article Snippet: ECG data analyses were performed off-line using Physiostat ECG analysis software, version 3.1 (Data Sciences International).

    Techniques:

    (A–F) EpA960/MCM and MCM littermates were given tamoxifen (TAM) at the same time. (A and D) H&E stain revealed that EpA960/MCM hearts were dilated compared with hearts of tamoxifen-treated MCM mice. Original magnification, ×2. (B and E) H&E stain revealed that induced EpA960/MCM hearts exhibited hypertrophied cardiomyocytes that focally contained pale, granular cytoplasm compared with MCM hearts. Original magnification, ×40. (C and F) Electron microscopy revealed hypertrophied myocytes with irregular nuclei (N) and abundant mitochondria (m) in EpA960/MCM hearts compared with MCM hearts. Original magnification, ×4,000. (G) Cardiac functional abnormalities in mice expressing EpA960(R) mRNA. Doppler ultrasound was performed before (Pre) and after (Post) tamoxifen administration on EpA960/MCM and MCM littermates and revealed both systolic and diastolic dysfunction in hearts expressing EpA960(R) mRNA. (H) ECG telemetry revealed progressive arrhythmias following induction of expanded CUG RNA expression. The length of the PR interval (thick bar) is indicated; thin bars represent 100 ms.

    Journal:

    Article Title: Elevation of RNA-binding protein CUGBP1 is an early event in an inducible heart-specific mouse model of myotonic dystrophy

    doi: 10.1172/JCI32308

    Figure Lengend Snippet: (A–F) EpA960/MCM and MCM littermates were given tamoxifen (TAM) at the same time. (A and D) H&E stain revealed that EpA960/MCM hearts were dilated compared with hearts of tamoxifen-treated MCM mice. Original magnification, ×2. (B and E) H&E stain revealed that induced EpA960/MCM hearts exhibited hypertrophied cardiomyocytes that focally contained pale, granular cytoplasm compared with MCM hearts. Original magnification, ×40. (C and F) Electron microscopy revealed hypertrophied myocytes with irregular nuclei (N) and abundant mitochondria (m) in EpA960/MCM hearts compared with MCM hearts. Original magnification, ×4,000. (G) Cardiac functional abnormalities in mice expressing EpA960(R) mRNA. Doppler ultrasound was performed before (Pre) and after (Post) tamoxifen administration on EpA960/MCM and MCM littermates and revealed both systolic and diastolic dysfunction in hearts expressing EpA960(R) mRNA. (H) ECG telemetry revealed progressive arrhythmias following induction of expanded CUG RNA expression. The length of the PR interval (thick bar) is indicated; thin bars represent 100 ms.

    Article Snippet: Data collection was performed using Dataquest software, and offline data analyses were performed using Physiostat ECG analysis software (version 3.1; Data Sciences International).

    Techniques: Staining, Electron Microscopy, Functional Assay, Expressing, RNA Expression